Licencia de Creative Commons Reconocimiento-NoComercial-CompartirIgual 4.0 InternacionalChang, Aileen Y.Lynch, RebeccaMartins, KarenEncinales, LilianaCadena Bonfanti, Andrés Á.Pacheco, NellyReid, St. PatrickLara Sarabia, Osvaldo E.González Torres, Henry J.Mejia Castillo, StellaBarrios Taborda, OnaldoAlarcon Gomez, MagdaGuerra Duran, BrendaMartinez Giraldo, VictorBenitez Ospino, AngélicaPorras, AlexandraMendoza, Alejandro R.Mantus, GraceLi, GuangzhaoPeng, JinKamalapathy, PriyankaAvendaño Echavez, Lil G.Dowd, Kimberly A.Rengifo-Pardo, MonicaBarraza, Pedro PabloJiménez Hernàndez, DennysGonzález Coba, AndrésDe La Hoz Mendoza, KatyaBethony, Jeffrey M.Simon, Gary L.2018-09-042018-09-042018-0822221751http://hdl.handle.net/20.500.12442/2237Zika virus infection has been associated with the development of a spectrum of neurologic disease including Guillain–Barré syndrome (GBS)1. GBS is an autoimmune disorder of the peripheral nervous system often triggered by a preceding infection. The mechanism of Zika-associated GBS (Z-GBS) and the long-term clinical course is unknown. The purpose of this study was to describe the 2-year clinical course of Z-GBS in order to provide further insights into disease pathogenesis and prognosis.engZika-VirusGuillain-Barre SyndromeChinkungunya infectionEpidemiologyInfectious disease controlRespiratory infectionsarticleinfo:eu-repo/semantics/openAccess